Up to this point the focus of this blog has centered on the causes of Type 1 DM. It is time to turn the page and start examining how a cure can be reached. I have been deliberating on how best to resolve this and it has become apparent that the inflammation and how the immune system fails to resolve it is paramount in making any inroads to being diabetes free.
Having been exposed to a lot of interesting alternative interventions, I have continually come back to several protocols devised by Walther H. Schmitt and Kerry McCord, two ICAK diplomates. These two gentlemen are Chiropractors who have created a comprehensive clinical protocol that treats all aspects of the body. I am interested to conduct the ‘bleach sniff test’, which is a screen for free radical pathology to see the results. In the ‘Quintessential Applications’ protocol it notes that symptoms of free radical pathology include autoimmune diseases. I am also interested to use ‘injury recall technique with offending agents’, which in this case would be foods, to the Neurolymphatic reflexes for my organs and glands. I hope that this too will achieve the result of decreasing any aberrant immune activity as well as decrease any inflammation. Although I have made it a habit to avoid any foods that I have determined provoke an immune response by the GI tract, I am curious to see if my situation improves by neurologically disassociating these foods from any inflammatory processes.
Also after reading this article it has become even more apparent that there is potential for many foods to provoke inflammatory responses, which I have to suspect contributes to the maintenance of the immune mediated destruction of the beta cells. I am planning on going back to yams and sweet potatoes in place of white/russet/yukon gold potatoes as a regular source of carbohydrates so as to avoid any potential inflammation on that front. It also seems that I need to focus on ensuring that I am getting my EFAs (essential fatty acids) and, perhaps more importantly, that my body is capable of using these fatty acids in the right places to curb inflammation and restore and repair cellular membranes. However, this will be addressed with the ‘bleach sniff test’ as it involves testing different nutrients involved with regulating inflammatory processes to see which ones negate a positive test, should that be the case.
Another source of inflammation is infection. In the case of T1D (Type 1 Diabetes) it would seem that enteroviruses are culprits worthy of investigation. Particularly interesting is the coxsackie (B) virus, which can affect the pancreas (meaning inflammation). Some suggest that it is what leads to T1D. Perhaps it is a contributing factor. It seems as though there are myriad potential causes but no definitive, singular cause. Cow’s milk seems to be a recurrent suggested cause but, it doesn’t effect the development of diabetes on it’s own. Some suggest that genes are the cause but, if you read this article or this article it suggesst that even when one identical twin has T1D, the other develops Type 1 at best 50% of the time. That would imply that genes are at best half of the reason someone develops T1D.
So, since this is my blog I will posit a theory: Type 1 Diabetes Mellitus begins with the development of a leaky gut. With the degradation of the integrity of the intestinal walls, immune protection against foreign invaders is compromised and, with digestion impaired the semi or undigested food passes into the blood stream, triggering immune responses that compound the situation. This amounts to considerable inflammation/oxidative tissue damage. If there is a leaky gut, it may have been provoked (at least, in part) by a lack of antioxidants which mean good fats and a number of nuts, vegetables and fruits. Or, the condition may have been exacerbated by a lack of antioxidants, which will ultimately lead to an ongoing cycle of oxidative tissue damage. Plus, if previously digested proteins (i.e bovine serum albumin) are now no longer being properly digested and the immune system is overburdened, it may, because of the similarity in conformations of the beta cells and BSA, resort to destroying the beta cells in error in an attempt to control the situation. And/or, an enterovirus affects the pancreas and the free radicals released by the immune system wind up damaging the beta cells. (There is also the gene aspect but, I will leave that aside for now.) This makes sense to me because the leaky gut seems to be the tie that binds between many autoimmune disease like MS, T1D, Crohn’s, Celiac disease and others. If one considers that the GI tract has the enteric nervous system, which has been likened to a second brain it becomes apparent that the body’s response pathogenesis and/or being bombarded with foods that it is sensitive/allergic to, the immune system may respond in unpredictable and complex ways.